我想問下有了iga的話,我應該eat什麼food??

Nephropathy - IgA; Berger's disease

Definition

IgA nephropathy is a kidney disorder characterized by blood in the urine. It is caused by inflammation of internal kidney structures and the deposit of IgA antibodies in kidney tissue.

Causes, incidence, and risk factors

IgA nephropathy (Berger's disease) is a form of mesangial proliferative nephritis. Structures in the kidney called glomeruli become inflamed, and a type of antibody called IgA deposits in the kidney. The disorder can appear as acute, rapidly progressive, or chronic glomerulonephritis, or as visible or microscopic blood in the urine.

IgA nephropathy usually is discovered after one or more episodes of dark or bloody urine in a person with no other symptoms of kidney disorder. Bloody urine may begin during or soon after a respiratory infection. Acute nephritic syndrome or nephrotic syndrome (groups of symptoms associated with decreased kidney functioning) may result.

Risk factors include having a personal or family history of IgA nephropathy or Henoch Schonlein purpura (a form of vasculitis that affects many parts of the body, and may cause a kidney lesion that is identical to the lesion of Berger's disease).

IgA nephropathy can occur in persons of all ages, but most often affects males in their teens to late 30s.

Symptoms

Changes in color of urine - bloody, dark, brown, or rust colored
Repeated episodes of dark or bloody urine

Signs and tests

There are no specific changes seen during a physical examination. Occasionally, blood pressure may be elevated or swelling of the body may be present.

Urinalysis shows blood and red cell casts in the urine, and may also show protein and fats.
Kidney biopsy shows deposits of IgA antibodies and confirms the diagnosis.
Urine immunoelectrophoresis shows antibodies to IgA.

Treatment

The goal of treatment is to relieve symptoms and prevent or delay chronic renal failure

Antihypertensive (high blood pressure) medications and diuretics may be given to control high blood pressure and edema. Control of blood pressure is the most important measure to delay renal (kidney) damage. Sodium (salt) and fluids may be restricted to control swelling. A low- to moderate-protein diet may be recommended in some cases.

The use of ACE inhibitors is key to preventing this disease from getting worse. Corticosteroids, other immunosuppressive drugs, and fish oil have also been used to treat this disorder.

Support Groups

For additional information and support, see the IgA Nephropathy Support Network website (www.igansupport.org).

Expectations (prognosis)

IgA nephropathy progresses slowly. In many cases, it does not progress at all. High blood pressure, large quantities of protein in the urine, and increased BUN or creatinine levels (blood tests that reflect kidney functioning) indicate a higher risk for progression of the disorder.

About 25% of people with IgA nephropathy develop end-stage kidney failure within about 25 years.

Complications

Chronic kidney failure
End-stage kidney disease

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The major food sources of protein are:

Highest Amount of Protein

Meat, fish, poultry, eggs
Milk, yogurt, cheese, peanut butter

Small Amount of Protein

Vegetables
Starches such as breads, cereals, and pasta (the protein in these foods is of poorer but still good quality)

Trace Amount of Protein

Fruits
Fats
Sugars

I hope you find these information useful

我個人經驗，全素食後就好少病發，不知不覺都十五年了，只要介口，什麼肉類都不吃，如果持之以恆，少了病發後，我就慢慢開始吃海鮮，魚，我暫時都okay. 但人人唔同，這是我的個人經驗。