✔ 最佳答案
To the poster above me - I don't know how 'professional' you are as you claimed to be but the answer doesn't look too professional to me (dare I say at all)! I don't think if a person is depressed or slow brain funcation can lead to one's death! You get upset and depressed every now and then - it looks like you're still alive and well! Please - do not draw simple and naive conclusions like that and claimed to be a 'professional' contribution to the question. An answer like that is simply misleading.
為什麼安眠藥 灸能跟酒一起渴?
Sleeping pills are a type of sedatives. Some of the major active compounds of sleeping pills are barbiturates or benzodiazepines. These compounds are known to bind to the GABA receptors in our brain. Here's a small introduction to GABA -
GABA is a neurotransmitter in our brain. It is what's called an 'inhibitory neurotransmitter'. It is simply a chemical (a simple amino acid) that when it is released and bind to the receptor, a person's actions are slowed down.
Think about the binding mecahism like a piece of Lego block and a another piece of Lego. If the shapes fit together, then there will be the slowing down action - if the pieces don't fit, then no actions.
Just as there is a 'slowing down' mechanism, there is also a 'hyperactive action'. This is the job of another neurotransmitter dopamine. So when there's GABA, there's dopamine.
Now on a GABA receptor, not only GABA can bind to it but also sedatives such as barbiturates and benzodiazepines. Just like there are 2 to 3 pieces of Lego that can fit together. So when those chemicals bind to the receptor, actions are slowed down and one of the results is sleepiness. Sciences are still trying to work out the exact mechanism of sleep - how does it work? what are the main triggers? etc. etc. Plus, the field of GABA and GABA receptors is still very young (about 30+ years), a lot still needs to be done in order to understand more about the field. It'll probably take another 50 years at the current rate before anything new will happen (in my opinion).
With the case of alcohol, sciencists have also found that alcohol can bind to GABA receptors. However, we don't know where exactly it binds but the result of that is the same as the binding of barbiturates and benzodiazepines - ie. slowed down of actions.
You might asked 'What's casuing the slowing down then?' To put it simply - when GABA receptors are activated (something binding to it), something called a 'chloride channel' opens, allowing -ve chloride ions to entre the channel. This will change the current of the cell membrane and in this case, actions are slowed down. This is a rather complicated topic that can't be explained in a few words but you can look at 'action potential' on the web for more details. One bad thing about opening of these ion channels is that they can 'die' after a while - ie. they cannot be close because they have been overworked. This is called 'desensitised' - so you can imagine the person's action is paralysed. So alcohol can contribute to that result.
Of course, there will also be the possible interactions between drug compounds and alcohol during metabolism or side reactions that will cause liver damage and death. Don't forger drugs need to be metabolised and some of the metabolites are toxic.
So I have basically answered both 還有安眠藥成份是什麼?? and 為何能令人安睡?? above. It's something that really can't be explain in a few words and lead to many other fields including pharmacology and physiology.
I hope I have helped a bit - hope you can understand what I said.